[buggerlugs]

Explaining The Covid 19 Virus

THE BEST EXPLANATION OF COVID 19
Elizabeth Hallam
Yesterday at 7:58 AM ·
This was shared by Amanda Howson RNand tells us why this bug is NOT like the
influenza virus influenza.
This was posted by a friend in Pathology. Thought it explained things really well.
Oh OT, ED RN, NSW.
I was asked today what is the difference between a influenza and COVID19.... can
we just have one day free?! Anyway here is the info to help you all,
“Feeling confused as to why Coronavirus is a bigger deal than Seasonal flu? Here it
is in a nutshell. I hope this helps. Feel free to share this with others who don’t
understand.”
It has to do with RNA sequencing...i.e. genetics.
Seasonal flu is an “all human virus”. The DNA/RNA chains that make up the virus are
recognized by the human immune system. This means that your body has some
immunity to it before it comes around each year. You get immunity two
ways...through exposure to a virus, or by getting a flu shot.
Novel viruses come from animals. The WHO tracks novel viruses in animals
(sometimes for years watching for mutations). Usually these viruses only transfer
from animal to animal (pigs in the case of H1N1, birds in the case of the Spanish flu).
But once one of these animal viruses mutates and starts to transfer from animals to
humans...then it’s a problem. Why? Because we have no natural or acquired
immunity. The RNA sequencing of the genes inside the virus isn’t human, and the
human immune system doesn’t recognize it, so we can’t fight it off.
Now...sometimes, the mutation only allows transfer from animal to human. For years
it’s only transmission is from an infected animal to a human before it finally mutates
so that it can now transfer human to human. Once that happens, we have a new
contagion phase. And depending on the fashion of this new mutation, that’s what
decides how contagious, or how deadly, it’s going to be.
H1N1 was deadly, but it did not mutate in a way that was as deadly as the Spanish
flu. Its RNA was slower to mutate and it attacked its host differently, too.
Fast forward.
Now, here comes this Coronavirus. It existed in animals only, for nobody knows how
long. But one day, at an animal market in Wuhan China, in December 2019, it
mutated and made the jump from animal to people.

At first, only animals could give it to a person. But here is the scary part. In just TWO
WEEKS, it mutated again and gained the ability to jump from human to human.
Scientists call this quick ability, “slippery”.
This Coronavirus, not being in any form a “human” virus (whereas we would all have
some natural or acquired immunity), took off like a rocket. And this was because
humans have no known immunity...doctors have no known medicines for it.
And it just so happens that this particular mutated animal virus changed itself in such
a way the way that it causes great damage to human lungs.
That’s why Coronavirus is different from seasonal flu, or H1N1, or any other type of
influenza...this one is slippery AF. And it’s a lung eater. And, it’s already mutated
AGAIN, so that we now have two strains to deal with, strain S and strain L...which
makes it twice as hard to develop a vaccine.
We really have no tools in our shed for this. History has shown that fast and
immediate closings of public places has helped in the past pandemics. Philadelphia
and Baltimore were reluctant to close events in 1918 and they were the hardest hit in
the US during the Spanish Flu.
Factoid: Henry VIII stayed in his room and allowed no one near him till the Black
Plague passed (honestly, I understand him so much better now). Just like us, he had
no tools in his shed, except social isolation.
And let me end by saying...right now it’s hitting older folks harder...but this genome is
so slippery, if it mutates again (and it will), who is to say what it will do next.
!!!! flatten the curve.
Stay home folks. And share this to those that just are not catching on."